Morphine Postconditioning Protects Against Reperfusion Injury: the Role of Protein Kinase C-Epsilon, Extracellular Signal-Regulated Kinase 1/2 and Mitochondrial Permeability Transition Pores. [electronic resource]
Producer: 20170207Description: 1930-1940 p. digitalISSN:- 1421-9778
- Animals
- Creatine Kinase, MB Form -- metabolism
- Cytochromes c -- metabolism
- Gene Expression Regulation
- Ischemic Postconditioning -- methods
- Male
- Mitochondrial Membrane Transport Proteins -- genetics
- Mitochondrial Permeability Transition Pore
- Mitogen-Activated Protein Kinase 1 -- genetics
- Mitogen-Activated Protein Kinase 3 -- genetics
- Morphine -- antagonists & inhibitors
- Myocardial Reperfusion Injury -- genetics
- Organ Culture Techniques
- Peptide Fragments -- pharmacology
- Phosphorylation -- drug effects
- Protein Kinase C-epsilon -- genetics
- Protein Transport -- drug effects
- Rats
- Rats, Sprague-Dawley
- Signal Transduction
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Publication Type: Journal Article
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