Clk1 deficiency promotes neuroinflammation and subsequent dopaminergic cell death through regulation of microglial metabolic reprogramming. [electronic resource]
Producer: 20180816Description: 206-219 p. digitalISSN:- 1090-2139
- Animals
- Cell Death -- genetics
- Cells, Cultured
- Dopamine -- metabolism
- Dopaminergic Neurons -- metabolism
- Inflammation -- metabolism
- Lipopolysaccharides -- pharmacology
- Mice, Knockout
- Microglia -- metabolism
- Nerve Degeneration -- metabolism
- Protein Serine-Threonine Kinases -- deficiency
- Protein-Tyrosine Kinases -- deficiency
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Publication Type: Journal Article
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