The m-AAA Protease Associated with Neurodegeneration Limits MCU Activity in Mitochondria. [electronic resource]
Producer: 20170829Description: 148-162 p. digitalISSN:- 1097-4164
- ATP-Dependent Proteases -- genetics
- ATPases Associated with Diverse Cellular Activities
- Animals
- Calcium -- metabolism
- Calcium Channels -- genetics
- Calcium-Calmodulin-Dependent Protein Kinase Type 2 -- genetics
- Cell Death
- Cerebellum -- metabolism
- Corpus Striatum -- metabolism
- Gene Expression Regulation
- HEK293 Cells
- Hippocampus -- metabolism
- Homeostasis -- genetics
- Humans
- Ion Transport
- Metalloendopeptidases -- deficiency
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Mitochondria -- metabolism
- Mitochondrial Membrane Transport Proteins -- genetics
- Mitochondrial Permeability Transition Pore
- Neurons -- metabolism
- Protein Interaction Mapping
- Signal Transduction
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Publication Type: Journal Article
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