Helicobacter pylori-induced chronic inflammation causes telomere shortening of gastric mucosa by promoting PARP-1-mediated non-homologous end joining of DNA. [electronic resource]
Producer: 20170523Description: 90-8 p. digitalISSN:- 1096-0384
- Adult
- Chronic Disease
- DNA -- chemistry
- DNA Damage
- DNA End-Joining Repair
- Female
- Gastric Mucosa -- metabolism
- Gastritis -- metabolism
- Helicobacter Infections -- drug therapy
- Helicobacter pylori -- pathogenicity
- Histones -- chemistry
- Humans
- Hydrogen Peroxide -- chemistry
- Inflammation -- microbiology
- Male
- Middle Aged
- NF-kappa B -- metabolism
- Oxidative Stress
- Oxygen -- chemistry
- Poly (ADP-Ribose) Polymerase-1 -- metabolism
- RNA, Small Interfering -- metabolism
- Telomere -- chemistry
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Publication Type: Journal Article
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