The von Hippel-Lindau Chuvash mutation in mice alters cardiac substrate and high-energy phosphate metabolism. [electronic resource]
Producer: 20170626Description: H759-67 p. digitalISSN:- 1522-1539
- Adenosine Triphosphate -- metabolism
- Animals
- Apoptosis Regulatory Proteins
- Aryl Hydrocarbon Receptor Nuclear Translocator
- Basic Helix-Loop-Helix Transcription Factors
- Carbon Isotopes
- Cardiotonic Agents -- pharmacology
- Disease Models, Animal
- Glucose -- metabolism
- Glycolysis
- Heart -- drug effects
- Hypertension, Pulmonary -- diagnostic imaging
- Hypertrophy, Right Ventricular -- diagnostic imaging
- Hypoxia-Inducible Factor 1, alpha Subunit
- Isolated Heart Preparation
- Isoproterenol -- pharmacology
- L-Lactate Dehydrogenase -- metabolism
- Magnetic Resonance Imaging
- Magnetic Resonance Spectroscopy
- Mice
- Mutation
- Myocardium -- metabolism
- Phosphates -- metabolism
- Phosphocreatine -- metabolism
- Polycythemia -- congenital
- Pyruvic Acid -- metabolism
- Repressor Proteins
- Stroke Volume
- Transcription Factors
- Tritium
- Von Hippel-Lindau Tumor Suppressor Protein -- genetics
No physical items for this record
Publication Type: Journal Article
There are no comments on this title.
Log in to your account to post a comment.