APA

Hullinger R., Li M., Wang J., Peng Y., Dowell J. A., Bomba-Warczak E., Mitchell H. A., Burger C., Chapman E. R., Denu J. M., Li L. & Puglielli L. (20170728). Increased expression of AT-1/SLC33A1 causes an autistic-like phenotype in mice by affecting dendritic branching and spine formation. : The Journal of experimental medicine.

Chicago

Hullinger Rikki, Li Mi, Wang Jingxin, Peng Yajing, Dowell James A, Bomba-Warczak Ewa, Mitchell Heather A, Burger Corinna, Chapman Edwin R, Denu John M, Li Lingjun and Puglielli Luigi. 20170728. Increased expression of AT-1/SLC33A1 causes an autistic-like phenotype in mice by affecting dendritic branching and spine formation. : The Journal of experimental medicine.

Harvard

Hullinger R., Li M., Wang J., Peng Y., Dowell J. A., Bomba-Warczak E., Mitchell H. A., Burger C., Chapman E. R., Denu J. M., Li L. and Puglielli L. (20170728). Increased expression of AT-1/SLC33A1 causes an autistic-like phenotype in mice by affecting dendritic branching and spine formation. : The Journal of experimental medicine.

MLA

Hullinger Rikki, Li Mi, Wang Jingxin, Peng Yajing, Dowell James A, Bomba-Warczak Ewa, Mitchell Heather A, Burger Corinna, Chapman Edwin R, Denu John M, Li Lingjun and Puglielli Luigi. Increased expression of AT-1/SLC33A1 causes an autistic-like phenotype in mice by affecting dendritic branching and spine formation. : The Journal of experimental medicine. 20170728.