Angiopoietin-1 attenuates angiotensin II-induced ER stress in glomerular endothelial cells via a Tie2 receptor/ERK1/2-p38 MAPK-dependent mechanism. [electronic resource]
Producer: 20171006Description: 118-32 p. digitalISSN:- 1872-8057
- Adenoviridae -- metabolism
- Angiopoietin-1 -- pharmacology
- Angiotensin II -- adverse effects
- Animals
- Apoptosis -- drug effects
- Biomarkers -- metabolism
- Cell Line
- Cell Movement -- drug effects
- Cells, Cultured
- Endoplasmic Reticulum Chaperone BiP
- Endoplasmic Reticulum Stress -- drug effects
- Endothelial Cells -- enzymology
- Heat-Shock Proteins -- metabolism
- Kidney Glomerulus -- pathology
- MAP Kinase Signaling System -- drug effects
- Mice
- Nitric Oxide -- metabolism
- Protein Kinase Inhibitors -- pharmacology
- RNA, Messenger -- genetics
- Receptor, TIE-2 -- metabolism
- Transcription Factor CHOP -- metabolism
- p38 Mitogen-Activated Protein Kinases -- metabolism
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Publication Type: Journal Article
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