The renin-angiotensin system and its vasoactive metabolite angiotensin-(1-7) in the mechanism of the healing of preexisting gastric ulcers. The involvement of Mas receptors, nitric oxide, prostaglandins and proinflammatory cytokines. [electronic resource]
Producer: 20161214Description: 75-91 p. digitalISSN:- 1899-1505
- Angiotensin I -- metabolism
- Angiotensin II -- metabolism
- Angiotensin-Converting Enzyme Inhibitors -- pharmacology
- Animals
- Cytokines -- metabolism
- Enzyme Inhibitors -- pharmacology
- Gastric Mucosa -- drug effects
- Imidazoles -- pharmacology
- Indomethacin -- pharmacology
- Interleukin-1beta -- metabolism
- Lisinopril -- pharmacology
- Losartan -- pharmacology
- Male
- NG-Nitroarginine Methyl Ester -- pharmacology
- Nitric Oxide -- metabolism
- Nitric Oxide Synthase -- metabolism
- Peptide Fragments -- metabolism
- Prostaglandins -- metabolism
- Proto-Oncogene Mas
- Proto-Oncogene Proteins -- metabolism
- Rats
- Rats, Wistar
- Receptors, G-Protein-Coupled -- metabolism
- Renin-Angiotensin System -- drug effects
- Stomach Ulcer -- metabolism
- Superoxide Dismutase -- metabolism
- Tumor Necrosis Factor-alpha -- metabolism
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.