Prolyl hydroxylase domain 2 deficiency promotes skeletal muscle fiber-type transition via a calcineurin/NFATc1-dependent pathway. [electronic resource]
Producer: 20160928Description: 5 p. digitalISSN:- 2044-5040
- Animals
- Calcineurin -- metabolism
- Calcineurin Inhibitors -- pharmacology
- Capillaries -- metabolism
- Cell Hypoxia
- Cell Line
- Genotype
- Hydroxylation
- Hypoxia-Inducible Factor 1, alpha Subunit -- metabolism
- Hypoxia-Inducible Factor-Proline Dioxygenases -- deficiency
- Mice, Knockout
- Muscle Fibers, Slow-Twitch -- drug effects
- Muscle, Skeletal -- blood supply
- NFATC Transcription Factors -- metabolism
- Neovascularization, Physiologic
- Phenotype
- Protein Stability
- Signal Transduction -- drug effects
- Tacrolimus -- pharmacology
- Time Factors
- Vascular Endothelial Growth Factor A -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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