Functional Genomic Screening Reveals Splicing of the EWS-FLI1 Fusion Transcript as a Vulnerability in Ewing Sarcoma. [electronic resource]
Producer: 20161020Description: 598-610 p. digitalISSN:- 2211-1247
- Base Sequence
- Binding Sites
- Calmodulin-Binding Proteins -- antagonists & inhibitors
- Cell Line, Tumor
- Cell Survival
- Exons
- Gene Expression Regulation, Neoplastic
- Heterogeneous-Nuclear Ribonucleoprotein Group F-H -- antagonists & inhibitors
- Humans
- Microfilament Proteins -- antagonists & inhibitors
- Oncogene Proteins, Fusion -- antagonists & inhibitors
- Phosphoproteins -- antagonists & inhibitors
- Proto-Oncogene Protein c-fli-1 -- antagonists & inhibitors
- RNA Interference
- RNA Precursors -- metabolism
- RNA Splicing
- RNA Splicing Factors
- RNA, Small Interfering -- metabolism
- RNA-Binding Protein EWS -- antagonists & inhibitors
- RNA-Binding Proteins -- antagonists & inhibitors
- Receptors, Cytoplasmic and Nuclear -- antagonists & inhibitors
- Ribonucleoprotein, U2 Small Nuclear -- antagonists & inhibitors
- Sarcoma, Ewing -- pathology
- Trans-Activators
- Transcription Factors -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't
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