Neuronostatin acts via GPR107 to increase cAMP-independent PKA phosphorylation and proglucagon mRNA accumulation in pancreatic α-cells. [electronic resource]
Producer: 20160502Description: R143-55 p. digitalISSN:- 1522-1490
- Animals
- Cell Line
- Cyclic AMP -- metabolism
- Cyclic AMP-Dependent Protein Kinases -- metabolism
- Glucagon-Secreting Cells -- drug effects
- Humans
- Male
- Mice
- Peptide Fragments -- metabolism
- Peptide Hormones -- metabolism
- Phosphorylation
- Proglucagon -- genetics
- RNA Interference
- RNA, Messenger -- genetics
- Rats, Sprague-Dawley
- Receptors, G-Protein-Coupled -- agonists
- Signal Transduction -- drug effects
- Somatostatin -- metabolism
- Transfection
- Up-Regulation
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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