HMGB1-Driven Inflammation and Intimal Hyperplasia After Arterial Injury Involves Cell-Specific Actions Mediated by TLR4. [electronic resource]
Producer: 20160317Description: 2579-93 p. digitalISSN:- 1524-4636
- Adaptor Proteins, Vesicular Transport -- metabolism
- Animals
- Carotid Arteries -- metabolism
- Carotid Artery Injuries -- genetics
- Cell Movement
- Cell Proliferation
- Cells, Cultured
- Chemotaxis, Leukocyte
- Cytokines -- metabolism
- Disease Models, Animal
- HMGB1 Protein -- deficiency
- Humans
- Hyperplasia
- Inflammation Mediators -- metabolism
- Macrophages, Peritoneal -- metabolism
- Mice, Inbred C57BL
- Mice, Knockout
- Myeloid Differentiation Factor 88 -- genetics
- Myocytes, Smooth Muscle -- metabolism
- Neointima
- Signal Transduction
- Time Factors
- Toll-Like Receptor 4 -- deficiency
- Vascular System Injuries -- genetics
- Vasculitis -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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