Clinically relevant concentrations of lidocaine and ropivacaine inhibit TNFα-induced invasion of lung adenocarcinoma cells in vitro by blocking the activation of Akt and focal adhesion kinase. [electronic resource]
Producer: 20160125Description: 784-91 p. digitalISSN:- 1471-6771
- Adenocarcinoma -- metabolism
- Adenocarcinoma of Lung
- Amides -- pharmacology
- Anesthetics, Local -- pharmacology
- Caveolin 1 -- metabolism
- Cell Movement -- drug effects
- Drug Evaluation, Preclinical -- methods
- Enzyme Activation -- drug effects
- Focal Adhesion Protein-Tyrosine Kinases -- metabolism
- Humans
- Lidocaine -- pharmacology
- Lung Neoplasms -- metabolism
- Matrix Metalloproteinase 9 -- metabolism
- Neoplasm Invasiveness
- Neoplasm Proteins -- metabolism
- Phosphorylation -- drug effects
- Proto-Oncogene Proteins c-akt -- metabolism
- Ropivacaine
- Tumor Cells, Cultured
- Tumor Necrosis Factor-alpha -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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