Sevoflurane post-conditioning reduces rat myocardial ischemia reperfusion injury through an increase in NOS and a decrease in phopshorylated NHE1 levels. [electronic resource]
Producer: 20160912Description: 1529-37 p. digitalISSN:- 1791-244X
- Anesthetics, Inhalation -- pharmacology
- Animals
- Autophagy -- drug effects
- Blotting, Western
- Cardiotonic Agents -- pharmacology
- Caspase 3 -- metabolism
- Enzyme Inhibitors -- pharmacology
- Heart -- drug effects
- In Vitro Techniques
- Ischemic Postconditioning -- methods
- Male
- Methyl Ethers -- pharmacology
- Microscopy, Electron, Transmission
- Microtubule-Associated Proteins -- metabolism
- Mitochondrial Membrane Transport Proteins -- metabolism
- Mitochondrial Permeability Transition Pore
- Myocardial Infarction -- genetics
- Myocardial Reperfusion Injury -- genetics
- Myocardium -- metabolism
- NG-Nitroarginine Methyl Ester -- pharmacology
- Nitric Oxide Synthase -- antagonists & inhibitors
- Phagosomes -- drug effects
- Phosphorylation -- drug effects
- Rats, Sprague-Dawley
- Reverse Transcriptase Polymerase Chain Reaction
- Sevoflurane
- Sodium-Hydrogen Exchanger 1
- Sodium-Hydrogen Exchangers -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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