Fli1 deficiency contributes to the downregulation of endothelial protein C receptor in systemic sclerosis: a possible role in prothrombotic conditions. [electronic resource]
Producer: 20161214Description: 338-47 p. digitalISSN:- 1365-2133
- Adult
- Aged
- Analysis of Variance
- Animals
- Antigens, CD -- physiology
- Bleomycin -- pharmacology
- Bosentan
- Case-Control Studies
- Cells, Cultured
- Down-Regulation -- physiology
- Endothelial Cells -- metabolism
- Endothelial Protein C Receptor
- Endothelin Receptor Antagonists -- pharmacology
- Endothelium, Vascular -- metabolism
- Enzyme-Linked Immunosorbent Assay
- Female
- Fibrinolysin -- drug effects
- Humans
- Male
- Mice
- Microvessels -- metabolism
- Middle Aged
- Proto-Oncogene Protein c-fli-1 -- deficiency
- Receptors, Cell Surface -- physiology
- Scleroderma, Systemic -- blood
- Sulfonamides -- pharmacology
- Thrombophilia -- etiology
- alpha-2-Antiplasmin -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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