ΔNp63α Promotes Breast Cancer Cell Motility through the Selective Activation of Components of the Epithelial-to-Mesenchymal Transition Program. [electronic resource]
Producer: 20160104Description: 3925-35 p. digitalISSN:- 1538-7445
- Animals
- Breast Neoplasms -- mortality
- Cadherins -- biosynthesis
- Carcinoma, Ductal, Breast -- pathology
- Carcinoma, Intraductal, Noninfiltrating -- pathology
- Cell Movement
- Cell Transformation, Neoplastic -- pathology
- Disease Progression
- Epithelial Cells -- pathology
- Epithelial-Mesenchymal Transition -- physiology
- Female
- Gene Expression Regulation, Neoplastic
- Heterografts
- Humans
- Kaplan-Meier Estimate
- Mice
- Mice, Inbred NOD
- Mice, SCID
- MicroRNAs -- biosynthesis
- Neoplasm Invasiveness -- physiopathology
- Neoplasm Proteins -- physiology
- Proto-Oncogene Proteins -- biosynthesis
- RNA, Small Interfering -- pharmacology
- Receptor Protein-Tyrosine Kinases -- biosynthesis
- Signal Transduction -- physiology
- Snail Family Transcription Factors
- Transcription Factors -- biosynthesis
- Transfection
- Tumor Cells, Cultured
- Tumor Suppressor Proteins -- physiology
- Axl Receptor Tyrosine Kinase
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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