Deletion of angiotensin-converting enzyme 2 exacerbates renal inflammation and injury in apolipoprotein E-deficient mice through modulation of the nephrin and TNF-alpha-TNFRSF1A signaling. [electronic resource]
Producer: 20160328Description: 255 p. digitalISSN:- 1479-5876
- Angiotensin I -- metabolism
- Angiotensin II -- metabolism
- Angiotensin-Converting Enzyme 2
- Animals
- Apolipoproteins E -- deficiency
- Gene Deletion
- Humans
- Inflammation -- pathology
- Kidney -- drug effects
- Male
- Membrane Proteins -- metabolism
- Mice, Inbred C57BL
- Mice, Knockout
- Models, Biological
- NADPH Oxidase 4
- NADPH Oxidases -- metabolism
- Oxidative Stress -- drug effects
- Peptide Fragments -- metabolism
- Peptidyl-Dipeptidase A -- deficiency
- Real-Time Polymerase Chain Reaction
- Receptor, Angiotensin, Type 1 -- metabolism
- Receptors, Tumor Necrosis Factor, Type I -- metabolism
- Recombinant Proteins -- pharmacology
- Signal Transduction -- drug effects
- Superoxides -- metabolism
- Tumor Necrosis Factor-alpha -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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