CTRP3 attenuates post-infarct cardiac fibrosis by targeting Smad3 activation and inhibiting myofibroblast differentiation. [electronic resource]
Producer: 20160909Description: 1311-25 p. digitalISSN:- 1432-1440
- AMP-Activated Protein Kinases -- metabolism
- Adenoviridae -- genetics
- Adipokines -- genetics
- Animals
- Cardiomegaly -- etiology
- Cell Differentiation -- drug effects
- Cells, Cultured
- Disease Models, Animal
- E1A-Associated p300 Protein -- metabolism
- Fibrosis
- Gene Expression
- Gene Expression Regulation -- drug effects
- Gene Knockdown Techniques
- Gene Transfer Techniques
- Genetic Vectors -- administration & dosage
- Male
- Myocardial Infarction -- complications
- Myofibroblasts -- cytology
- Phosphorylation
- Protein Binding
- Protein Transport
- Proto-Oncogene Proteins c-akt -- metabolism
- Rats
- Smad3 Protein -- metabolism
- Transduction, Genetic
- Transforming Growth Factor beta1 -- metabolism
- Ventricular Remodeling -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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