Huntington disease iPSCs show early molecular changes in intracellular signaling, the expression of oxidative stress proteins and the p53 pathway. [electronic resource]
Producer: 20160726Description: 1047-57 p. digitalISSN:- 1754-8411
- Animals
- Base Sequence
- Cell Differentiation
- Child
- Disease Models, Animal
- Embryonic Stem Cells -- cytology
- Enzyme Activation
- Humans
- Huntingtin Protein
- Huntington Disease -- metabolism
- Induced Pluripotent Stem Cells -- cytology
- MAP Kinase Signaling System
- Mice
- Molecular Sequence Data
- Mutation
- Nerve Tissue Proteins -- genetics
- Nuclear Proteins -- genetics
- Oxidative Stress
- Phosphorylation
- Signal Transduction
- Superoxide Dismutase -- metabolism
- Superoxide Dismutase-1
- Tumor Suppressor Protein p53 -- metabolism
- Wnt Proteins -- metabolism
- Young Adult
- beta Catenin -- metabolism
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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