APA

Parker M. J., Fryer A. E., Shears D. J., Lachlan K. L., McKee S. A., Magee A. C., Mohammed S., Vasudevan P. C., Park S., Benoit V., Lederer D., Maystadt I., Study D. & FitzPatrick D. R. (20160614). De novo, heterozygous, loss-of-function mutations in SYNGAP1 cause a syndromic form of intellectual disability. : American journal of medical genetics. Part A.

Chicago

Parker Michael J, Fryer Alan E, Shears Deborah J, Lachlan Katherine L, McKee Shane A, Magee Alex C, Mohammed Shehla, Vasudevan Pradeep C, Park Soo-Mi, Benoit Valérie, Lederer Damien, Maystadt Isabelle, Study Ddd and FitzPatrick David R. 20160614. De novo, heterozygous, loss-of-function mutations in SYNGAP1 cause a syndromic form of intellectual disability. : American journal of medical genetics. Part A.

Harvard

Parker M. J., Fryer A. E., Shears D. J., Lachlan K. L., McKee S. A., Magee A. C., Mohammed S., Vasudevan P. C., Park S., Benoit V., Lederer D., Maystadt I., Study D. and FitzPatrick D. R. (20160614). De novo, heterozygous, loss-of-function mutations in SYNGAP1 cause a syndromic form of intellectual disability. : American journal of medical genetics. Part A.

MLA

Parker Michael J, Fryer Alan E, Shears Deborah J, Lachlan Katherine L, McKee Shane A, Magee Alex C, Mohammed Shehla, Vasudevan Pradeep C, Park Soo-Mi, Benoit Valérie, Lederer Damien, Maystadt Isabelle, Study Ddd and FitzPatrick David R. De novo, heterozygous, loss-of-function mutations in SYNGAP1 cause a syndromic form of intellectual disability. : American journal of medical genetics. Part A. 20160614.