Serum amyloid A receptor blockade and incorporation into high-density lipoprotein modulates its pro-inflammatory and pro-thrombotic activities on vascular endothelial cells. [electronic resource]
Producer: 20160218Description: 11101-24 p. digitalISSN:- 1422-0067
- Apolipoprotein A-I -- metabolism
- Cells, Cultured
- Endothelial Cells -- cytology
- Enzyme-Linked Immunosorbent Assay
- Gene Expression Regulation -- drug effects
- Humans
- Immunohistochemistry
- Lipoproteins, HDL -- isolation & purification
- NF-kappa B -- genetics
- Peptides -- pharmacology
- Phosphatidylcholines -- pharmacology
- Receptor for Advanced Glycation End Products -- genetics
- Receptors, Formyl Peptide -- chemistry
- Receptors, Lipoxin -- chemistry
- Recombinant Proteins -- biosynthesis
- Serum Amyloid A Protein -- antagonists & inhibitors
- Thromboplastin -- genetics
- Tumor Necrosis Factor-alpha -- analysis
- Vascular Endothelial Growth Factor A -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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