Alterations in left ventricular function during intermittent hypoxia: Possible involvement of O-GlcNAc protein and MAPK signaling. [electronic resource]
Producer: 20160324Description: 150-8 p. digitalISSN:- 1791-244X
- Acetylglucosamine -- metabolism
- Animals
- Apoptosis -- physiology
- Blood Gas Analysis
- Blood Pressure -- physiology
- Calcium-Calmodulin-Dependent Protein Kinase Type 2 -- metabolism
- Cardiovascular Diseases -- physiopathology
- Caspase 3 -- metabolism
- Cell Hypoxia -- physiology
- Extracellular Signal-Regulated MAP Kinases -- metabolism
- In Situ Nick-End Labeling
- Interleukin-6 -- metabolism
- MAP Kinase Signaling System -- physiology
- Male
- Myocardium -- metabolism
- Myocytes, Cardiac -- metabolism
- N-Acetylglucosaminyltransferases -- metabolism
- NF-kappa B -- metabolism
- Phosphorylation
- Rats
- Rats, Wistar
- Sleep Apnea, Obstructive -- physiopathology
- Tumor Necrosis Factor-alpha -- metabolism
- Ventricular Function, Left -- physiology
- p38 Mitogen-Activated Protein Kinases -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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