Alpha-Linolenic Acid-Induced Increase in Neurogenesis is a Key Factor in the Improvement in the Passive Avoidance Task After Soman Exposure. [electronic resource]
Producer: 20160520Description: 251-69 p. digitalISSN:- 1559-1174
- Animals
- Antigens, Nuclear -- biosynthesis
- Atropine Derivatives -- therapeutic use
- Avoidance Learning -- drug effects
- Brain Damage, Chronic -- etiology
- Brain-Derived Neurotrophic Factor -- biosynthesis
- DNA Replication -- drug effects
- Diazepam -- therapeutic use
- Doublecortin Domain Proteins
- Electroshock
- Exploratory Behavior -- drug effects
- Hippocampus -- drug effects
- Male
- Mechanistic Target of Rapamycin Complex 1
- Microtubule-Associated Proteins -- biosynthesis
- Multiprotein Complexes -- antagonists & inhibitors
- Nerve Tissue Proteins -- biosynthesis
- Neurogenesis -- drug effects
- Neuropeptides -- biosynthesis
- Neuroprotective Agents -- antagonists & inhibitors
- Neurotoxins -- metabolism
- Oximes -- therapeutic use
- Proto-Oncogene Proteins c-akt -- biosynthesis
- Pyridinium Compounds -- therapeutic use
- Rats
- Rats, Sprague-Dawley
- Receptor, trkB -- physiology
- Signal Transduction -- drug effects
- Sirolimus -- pharmacology
- Soman -- toxicity
- Status Epilepticus -- chemically induced
- TOR Serine-Threonine Kinases -- antagonists & inhibitors
- alpha-Linolenic Acid -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
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