Hepatocyte-specific Bid depletion reduces tumor development by suppressing inflammation-related compensatory proliferation. [electronic resource]
Producer: 20160829Description: 1985-94 p. digitalISSN:- 1476-5403
- Animals
- BH3 Interacting Domain Death Agonist Protein -- genetics
- Carbon Tetrachloride -- toxicity
- Cell Proliferation -- drug effects
- Cells, Cultured
- Chemical and Drug Induced Liver Injury -- metabolism
- Cyclin D1 -- genetics
- Diethylnitrosamine -- toxicity
- Disease Models, Animal
- Hepatocytes -- cytology
- Hydrogen Peroxide -- toxicity
- Inflammation -- metabolism
- Interleukin-6 -- genetics
- Liver -- enzymology
- Liver Neoplasms -- etiology
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Mice, Transgenic
- Non-alcoholic Fatty Liver Disease -- metabolism
- Pyridines -- toxicity
- Tumor Necrosis Factor-alpha -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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