Cell clustering and delay/arrest in T-cell division implicate a novel mechanism of immune modulation by E. coli heat-labile enterotoxin B-subunits. [electronic resource]
Producer: 20150727Description: 150-62 p. digitalISSN:- 1090-2163
- Adjuvants, Immunologic -- pharmacology
- Animals
- Bacterial Toxins -- immunology
- Cell Cycle -- immunology
- Cell Line
- Cell Survival -- drug effects
- Enterotoxins -- immunology
- Escherichia coli Proteins -- immunology
- Histocompatibility Antigens Class II -- immunology
- Interleukin-2 -- immunology
- Macrophages -- cytology
- Mice
- Ovalbumin -- immunology
- Receptors, Cell Surface -- immunology
- T-Lymphocytes -- cytology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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