Prenatal nicotinic exposure augments cardiorespiratory responses to activation of bronchopulmonary C-fibers. [electronic resource]
Producer: 20160426Description: L922-30 p. digitalISSN:- 1522-1504
- Animals
- Apnea -- chemically induced
- Brain-Derived Neurotrophic Factor -- biosynthesis
- Bronchoalveolar Lavage Fluid -- chemistry
- Capsaicin -- pharmacology
- Diaphragm -- physiopathology
- Electromyography
- Female
- Ganglia -- cytology
- Humans
- Hypoxia
- Infant, Newborn
- Male
- Methacholine Chloride -- pharmacology
- Nerve Fibers, Unmyelinated -- drug effects
- Nerve Growth Factor -- biosynthesis
- Nicotine -- pharmacology
- Nodose Ganglion -- metabolism
- Pregnancy
- Prenatal Exposure Delayed Effects
- Random Allocation
- Rats
- Rats, Sprague-Dawley
- Receptor, trkA -- genetics
- Receptor, trkB -- genetics
- Recombinant Proteins
- Sensory System Agents -- pharmacology
- Smoking -- adverse effects
- Substance P -- immunology
- Sudden Infant Death -- etiology
- TRPV Cation Channels -- biosynthesis
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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