Mutated myocilin and heterozygous Sod2 deficiency act synergistically in a mouse model of open-angle glaucoma. [electronic resource]
Producer: 20160204Description: 3322-34 p. digitalISSN:- 1460-2083
- Actins -- metabolism
- Animals
- Apoptosis -- genetics
- Axons -- metabolism
- Cytoskeletal Proteins -- genetics
- Disease Models, Animal
- E-Selectin -- genetics
- Endoplasmic Reticulum Stress -- genetics
- Epistasis, Genetic
- Eye Proteins -- genetics
- Gene Expression
- Glaucoma, Open-Angle -- diagnosis
- Glycoproteins -- genetics
- Haploinsufficiency
- Heterozygote
- Intraocular Pressure
- Mice
- Mice, Knockout
- Mutation
- Optic Nerve Diseases -- genetics
- Phenotype
- Retinal Ganglion Cells -- metabolism
- Superoxide Dismutase -- deficiency
- Trabecular Meshwork -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Intramural
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