Propranolol induces regression of hemangioma cells via the down-regulation of the PI3K/Akt/eNOS/VEGF pathway. [electronic resource]
Producer: 20150911Description: 1414-20 p. digitalISSN:- 1545-5017
- Adrenergic beta-Antagonists -- therapeutic use
- Angiogenesis Inhibitors -- therapeutic use
- Cell Cycle Checkpoints -- drug effects
- Cell Proliferation -- drug effects
- Cells, Cultured
- Chromones -- pharmacology
- Cinnamates -- pharmacology
- Cyclin A2 -- biosynthesis
- Cyclin D2 -- biosynthesis
- Cyclin-Dependent Kinase Inhibitor p21 -- biosynthesis
- Endothelial Cells -- metabolism
- Hemangioma -- drug therapy
- Humans
- Infant
- Infant, Newborn
- Morpholines -- pharmacology
- NG-Nitroarginine Methyl Ester -- pharmacology
- Neoplasm Invasiveness -- pathology
- Neovascularization, Pathologic -- drug therapy
- Nitric Oxide -- biosynthesis
- Nitric Oxide Synthase Type III -- antagonists & inhibitors
- Norepinephrine -- pharmacology
- Pericytes -- metabolism
- Phosphatidylinositol 3-Kinases -- biosynthesis
- Phosphoinositide-3 Kinase Inhibitors
- Phosphorylation
- Proliferating Cell Nuclear Antigen -- biosynthesis
- Propranolol -- therapeutic use
- Proto-Oncogene Proteins c-akt -- biosynthesis
- Vascular Endothelial Growth Factor A -- antagonists & inhibitors
- Vascular Endothelial Growth Factor Receptor-1 -- pharmacology
- Vascular Endothelial Growth Factor Receptor-2 -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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