Overexpression of protein phosphatase 2A in a murine model of chronic myocardial infarction leads to increased adverse remodeling but restores the regulation of β-catenin by glycogen synthase kinase 3β. [electronic resource]
Producer: 20160301Description: 39-46 p. digitalISSN:- 1874-1754
- Animals
- Atrial Remodeling -- physiology
- Blotting, Western
- Cardiomyopathy, Dilated -- metabolism
- Cardiomyopathy, Hypertrophic -- metabolism
- Electrocardiography
- Gene Expression -- physiology
- Glycogen Synthase Kinase 3 -- metabolism
- Glycogen Synthase Kinase 3 beta
- Male
- Mice
- Mice, Transgenic
- Myocardial Infarction -- metabolism
- Protein Phosphatase 2 -- metabolism
- beta Catenin -- metabolism
No physical items for this record
Publication Type: Journal Article
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