Reduction of no-reflow and reperfusion injury with the synthetic 17β-aminoestrogen compound Prolame is associated with PI3K/Akt/eNOS signaling cascade. [electronic resource]
Producer: 20150914Description: 1 p. digitalISSN:- 1435-1803
- Animals
- Blotting, Western
- Disease Models, Animal
- Endothelial Cells -- drug effects
- Estrenes -- pharmacology
- Hemodynamics -- drug effects
- Humans
- Male
- Myocardial Infarction -- metabolism
- Myocardial Reperfusion Injury -- metabolism
- Nitric Oxide Synthase Type III -- metabolism
- No-Reflow Phenomenon -- metabolism
- Phosphatidylinositol 3-Kinases -- metabolism
- Proto-Oncogene Proteins c-akt -- metabolism
- Rats
- Rats, Wistar
- Signal Transduction -- drug effects
- Umbilical Veins
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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