Tyrosine kinase inhibition in leukemia induces an altered metabolic state sensitive to mitochondrial perturbations. [electronic resource]
Producer: 20160204Description: 1360-72 p. digitalISSN:- 1557-3265
- Animals
- Antineoplastic Combined Chemotherapy Protocols -- pharmacology
- Apoptosis -- drug effects
- Cell Line, Tumor
- Dihydrolipoyllysine-Residue Acetyltransferase -- genetics
- Disease Models, Animal
- Female
- Fusion Proteins, bcr-abl -- metabolism
- Humans
- Imatinib Mesylate -- pharmacology
- Ketone Oxidoreductases -- metabolism
- Leukemia, Myelogenous, Chronic, BCR-ABL Positive -- drug therapy
- Membrane Potential, Mitochondrial -- drug effects
- Mice
- Mice, Inbred C57BL
- Mice, Inbred NOD
- Mice, Knockout
- Mitochondria -- metabolism
- Mitochondrial Proteins -- genetics
- Mitochondrial Proton-Translocating ATPases -- antagonists & inhibitors
- Oligomycins -- pharmacology
- Protein Kinase Inhibitors -- pharmacology
- Protein-Tyrosine Kinases -- antagonists & inhibitors
- RNA Interference
- RNA, Small Interfering
- Superoxides -- metabolism
- fms-Like Tyrosine Kinase 3 -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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