Cross-talk between endoplasmic reticulum (ER) stress and the MEK/ERK pathway potentiates apoptosis in human triple negative breast carcinoma cells: role of a dihydropyrimidone, nifetepimine. [electronic resource]
Producer: 20150508Description: 3936-49 p. digitalISSN:- 1083-351X
- Animals
- Apoptosis -- drug effects
- Blotting, Western
- Cell Cycle -- drug effects
- Cell Proliferation -- drug effects
- Endoplasmic Reticulum Chaperone BiP
- Endoplasmic Reticulum Stress -- drug effects
- Female
- Heat-Shock Proteins -- antagonists & inhibitors
- Humans
- Immunoenzyme Techniques
- MAP Kinase Kinase Kinases -- genetics
- Male
- Mice
- Mice, Nude
- Mice, SCID
- Mitogen-Activated Protein Kinase 1 -- genetics
- Mitogen-Activated Protein Kinase 3 -- genetics
- Pyrimidinones -- pharmacology
- RNA, Messenger -- genetics
- RNA, Small Interfering -- genetics
- Real-Time Polymerase Chain Reaction
- Reverse Transcriptase Polymerase Chain Reaction
- Triple Negative Breast Neoplasms -- drug therapy
- Tumor Cells, Cultured
- Xenograft Model Antitumor Assays
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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