Inhibition of autophagy via activation of PI3K/Akt pathway contributes to the protection of ginsenoside Rb1 against neuronal death caused by ischemic insults. [electronic resource]
Producer: 20150622Description: 15426-42 p. digitalISSN:- 1422-0067
- Animals
- Apoptosis Regulatory Proteins -- biosynthesis
- Autophagy -- drug effects
- Beclin-1
- Brain Ischemia -- drug therapy
- CA1 Region, Hippocampal -- blood supply
- Cell Line, Tumor
- Chromones -- pharmacology
- Enzyme Activation -- drug effects
- Ginsenosides -- antagonists & inhibitors
- Glucose -- pharmacology
- Humans
- Male
- Microtubule-Associated Proteins -- biosynthesis
- Morpholines -- pharmacology
- Nerve Tissue Proteins -- biosynthesis
- Neuroblastoma -- pathology
- Neurons -- drug effects
- Neuroprotective Agents -- antagonists & inhibitors
- Oxygen -- pharmacology
- Phosphatidylinositol 3-Kinases -- physiology
- Phytotherapy
- Proto-Oncogene Proteins c-akt -- physiology
- Random Allocation
- Rats
- Rats, Wistar
- Reperfusion Injury -- drug therapy
- Signal Transduction -- drug effects
- Up-Regulation -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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