A prolyl-hydroxylase inhibitor, ethyl-3,4-dihydroxybenzoate, induces cell autophagy and apoptosis in esophageal squamous cell carcinoma cells via up-regulation of BNIP3 and N-myc downstream-regulated gene-1. [electronic resource]
Producer: 20150615Description: e107204 p. digitalISSN:- 1932-6203
- Apoptosis -- drug effects
- Autophagy -- drug effects
- Carcinoma, Squamous Cell -- drug therapy
- Cell Cycle Proteins -- biosynthesis
- Cell Line, Tumor
- Cell Membrane Permeability -- drug effects
- Cell Proliferation -- drug effects
- Cell Survival -- drug effects
- Esophageal Neoplasms -- drug therapy
- Esophageal Squamous Cell Carcinoma
- Humans
- Hydroxybenzoates -- pharmacology
- Intracellular Signaling Peptides and Proteins -- biosynthesis
- Membrane Proteins -- biosynthesis
- Mitochondrial Membranes -- physiology
- Prolyl-Hydroxylase Inhibitors -- pharmacology
- Proto-Oncogene Proteins -- biosynthesis
- RNA Interference
- RNA, Small Interfering
- S Phase Cell Cycle Checkpoints -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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