Constitutive activation of CaMKKα signaling is sufficient but not necessary for mTORC1 activation and growth in mouse skeletal muscle. [electronic resource]
Producer: 20141215Description: E686-94 p. digitalISSN:- 1522-1555
- Ablation Techniques -- adverse effects
- Animals
- Calcium Signaling -- drug effects
- Calcium-Calmodulin-Dependent Protein Kinase Kinase -- chemistry
- Crosses, Genetic
- Female
- Gene Expression Regulation -- drug effects
- Hypertrophy
- In Vitro Techniques
- Mechanistic Target of Rapamycin Complex 1
- Mice, Inbred C57BL
- Mice, Knockout
- Multiprotein Complexes -- agonists
- Muscle Development -- drug effects
- Muscle Proteins -- biosynthesis
- Muscle, Skeletal -- drug effects
- Phosphorylation -- drug effects
- Protein Kinase Inhibitors -- pharmacology
- Protein Processing, Post-Translational -- drug effects
- Ribosomal Protein S6 Kinases, 70-kDa -- metabolism
- TOR Serine-Threonine Kinases -- antagonists & inhibitors
- Up-Regulation -- drug effects
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural
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