Netrin-1 abrogates ischemia/reperfusion-induced cardiac mitochondrial dysfunction via nitric oxide-dependent attenuation of NOX4 activation and recoupling of NOS. [electronic resource]
Producer: 20150818Description: 174-85 p. digitalISSN:- 1095-8584
- Animals
- Cardiotonic Agents -- administration & dosage
- Disease Models, Animal
- Hydrogen Peroxide -- metabolism
- Male
- Mice
- Mice, Knockout
- Mitochondria, Heart -- drug effects
- Myocardial Reperfusion Injury -- genetics
- NADPH Oxidase 4
- NADPH Oxidases -- genetics
- Nerve Growth Factors -- administration & dosage
- Netrin-1
- Nitric Oxide -- metabolism
- Nitric Oxide Synthase -- metabolism
- Oxidative Stress
- Pterins -- pharmacology
- RNA Interference
- RNA, Small Interfering -- genetics
- Superoxides -- metabolism
- Tumor Suppressor Proteins -- administration & dosage
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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