Mechanisms involved in kinin-induced glioma cells proliferation: the role of ERK1/2 and PI3K/Akt pathways. [electronic resource]
Producer: 20151013Description: 235-44 p. digitalISSN:- 1573-7373
- Apoptosis
- Blotting, Western
- Bradykinin -- analogs & derivatives
- Bradykinin B1 Receptor Antagonists -- pharmacology
- Bradykinin B2 Receptor Antagonists -- pharmacology
- Cell Proliferation
- Dioxoles -- pharmacology
- Flow Cytometry
- Glioma -- drug therapy
- Humans
- Mitogen-Activated Protein Kinase 1 -- genetics
- Mitogen-Activated Protein Kinase 3 -- genetics
- Phosphatidylinositol 3-Kinases -- genetics
- Phosphorylation -- drug effects
- Proto-Oncogene Proteins c-akt -- genetics
- RNA, Messenger -- genetics
- Real-Time Polymerase Chain Reaction
- Receptor, Bradykinin B1 -- chemistry
- Receptor, Bradykinin B2 -- chemistry
- Reverse Transcriptase Polymerase Chain Reaction
- Sulfonamides -- pharmacology
- Tumor Cells, Cultured
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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