Silencing of JMJD2B induces cell apoptosis via mitochondria-mediated and death receptor-mediated pathway activation in colorectal cancer. [electronic resource]
Producer: 20150512Description: 491-500 p. digitalISSN:- 1751-2980
- Apoptosis -- genetics
- Caspases -- metabolism
- Colorectal Neoplasms -- genetics
- Cytochromes c -- metabolism
- Down-Regulation
- Endoplasmic Reticulum Chaperone BiP
- Endoplasmic Reticulum Stress -- physiology
- Gene Silencing
- Humans
- Jumonji Domain-Containing Histone Demethylases -- genetics
- Mitochondria -- physiology
- Neoplasm Proteins -- metabolism
- Proto-Oncogene Proteins c-bcl-2 -- biosynthesis
- RNA Interference
- Receptors, Death Domain -- physiology
- Tumor Cells, Cultured
- bcl-2 Homologous Antagonist-Killer Protein -- metabolism
- bcl-2-Associated X Protein -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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