The transcriptional responsiveness of LKB1 to STAT-mediated signaling is differentially modulated by prolactin in human breast cancer cells. [electronic resource]
Producer: 20141209Description: 415 p. digitalISSN:- 1471-2407
- AMP-Activated Protein Kinase Kinases
- Breast Neoplasms -- genetics
- Female
- Gene Expression Regulation, Neoplastic
- Humans
- MCF-7 Cells
- Prolactin -- metabolism
- Protein Serine-Threonine Kinases -- biosynthesis
- STAT3 Transcription Factor -- genetics
- STAT5 Transcription Factor -- genetics
- Signal Transduction
- Tumor Suppressor Proteins -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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