APOL1 risk variants enhance podocyte necrosis through compromising lysosomal membrane permeability. [electronic resource]
Producer: 20141006Description: F326-36 p. digitalISSN:- 1522-1466
- 4,4'-Diisothiocyanostilbene-2,2'-Disulfonic Acid -- pharmacology
- Actins -- metabolism
- Black or African American -- ethnology
- Apolipoprotein L1
- Apolipoproteins -- genetics
- Cells, Cultured
- Chloride Channels -- antagonists & inhibitors
- Chloroquine -- pharmacology
- Genetic Predisposition to Disease -- ethnology
- Genetic Variation -- genetics
- Glomerulosclerosis, Focal Segmental -- ethnology
- Humans
- Lipoproteins, HDL -- genetics
- Lysosomes -- physiology
- Necrosis -- physiopathology
- Permeability
- Podocytes -- drug effects
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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