Cathepsin K-mediated Notch1 activation contributes to neovascularization in response to hypoxia. [electronic resource]
Producer: 20151001Description: 3838 p. digitalISSN:- 2041-1723
- Animals
- Basic Helix-Loop-Helix Transcription Factors -- metabolism
- Capillaries -- pathology
- Cathepsin K -- genetics
- Cell Cycle Proteins -- metabolism
- Femoral Artery -- surgery
- Hindlimb -- blood supply
- Homeodomain Proteins -- metabolism
- Hypoxia -- genetics
- Ischemia
- Laser-Doppler Flowmetry
- Ligation
- Mice
- Mice, Knockout
- Muscle, Skeletal -- blood supply
- Neovascularization, Physiologic -- genetics
- Phosphorylation
- Proto-Oncogene Proteins c-akt -- metabolism
- RNA, Messenger -- metabolism
- Receptor, Notch1 -- metabolism
- Repressor Proteins -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction
- Transcription Factor HES-1
- Vascular Endothelial Growth Factor A -- metabolism
- Vascular Endothelial Growth Factor Receptor-1 -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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