Semaphorin 3A inactivation suppresses ischemia-reperfusion-induced inflammation and acute kidney injury. [electronic resource]
Producer: 20140908Description: F183-94 p. digitalISSN:- 1522-1466
- Acute Kidney Injury -- genetics
- Animals
- Apoptosis
- Apoptosis Regulatory Proteins -- metabolism
- Cell Line
- Cisplatin -- toxicity
- Cytokines -- metabolism
- Dendritic Cells -- metabolism
- Disease Models, Animal
- Epithelial Cells -- metabolism
- Inflammation Mediators -- metabolism
- Kidney -- drug effects
- Kidney Tubules -- metabolism
- Macrophages -- metabolism
- Mice
- Mice, Inbred C3H
- Mice, Inbred C57BL
- Mice, Knockout
- Mutation
- Nephritis -- genetics
- Neuropilin-1 -- metabolism
- Reperfusion Injury -- genetics
- Semaphorin-3A -- antagonists & inhibitors
- Toll-Like Receptor 4 -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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