VIP regulates CFTR membrane expression and function in Calu-3 cells by increasing its interaction with NHERF1 and P-ERM in a VPAC1- and PKCε-dependent manner. [electronic resource]
Producer: 20140828Description: C107-19 p. digitalISSN:- 1522-1563
- Adaptor Proteins, Signal Transducing
- Bronchi -- cytology
- Carrier Proteins -- metabolism
- Cell Line
- Cystic Fibrosis Transmembrane Conductance Regulator -- metabolism
- Cytoskeletal Proteins -- metabolism
- Epithelial Cells -- enzymology
- Golgi Matrix Proteins
- Humans
- Membrane Proteins -- metabolism
- Membrane Transport Proteins
- Microfilament Proteins -- metabolism
- Phosphoproteins -- genetics
- Phosphorylation
- Protein Binding
- Protein Kinase C-epsilon -- metabolism
- RNA Interference
- Receptors, Vasoactive Intestinal Polypeptide, Type I -- metabolism
- Signal Transduction
- Sodium-Hydrogen Exchangers -- genetics
- Time Factors
- Transfection
- Vasoactive Intestinal Peptide -- metabolism
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.