β2- and β3-adrenergic receptors drive COMT-dependent pain by increasing production of nitric oxide and cytokines. [electronic resource]
Producer: 20150212Description: 1346-1355 p. digitalISSN:- 1872-6623
- Adrenergic beta-2 Receptor Antagonists -- pharmacology
- Adrenergic beta-3 Receptor Antagonists -- pharmacology
- Animals
- Catechol O-Methyltransferase -- metabolism
- Catechol O-Methyltransferase Inhibitors -- pharmacology
- Chemokine CCL2 -- drug effects
- Cytokines -- metabolism
- Hyperalgesia -- metabolism
- Interleukin-1beta -- drug effects
- Interleukin-6 -- metabolism
- Male
- Nitric Oxide -- metabolism
- Nitric Oxide Synthase -- antagonists & inhibitors
- Rats
- Rats, Sprague-Dawley
- Receptors, Adrenergic, beta-2 -- metabolism
- Receptors, Adrenergic, beta-3 -- metabolism
- Tumor Necrosis Factor-alpha -- drug effects
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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