Astrocyte Kir4.1 ion channel deficits contribute to neuronal dysfunction in Huntington's disease model mice. [electronic resource]
Producer: 20140617Description: 694-703 p. digitalISSN:- 1546-1726
- Age Factors
- Animals
- Astrocytes -- metabolism
- Corpus Striatum -- physiopathology
- Disease Models, Animal
- Green Fluorescent Proteins -- genetics
- HEK293 Cells
- Hindlimb Suspension -- physiology
- Humans
- Huntingtin Protein
- Huntington Disease -- genetics
- In Vitro Techniques
- Locomotion -- genetics
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Nerve Tissue Proteins -- genetics
- Neurons -- metabolism
- Nuclear Proteins -- genetics
- Potassium Channels, Inwardly Rectifying -- deficiency
- Survival Analysis
- Trinucleotide Repeats -- genetics
- Kcnj10 Channel
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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