Defects in the C. elegans acyl-CoA synthase, acs-3, and nuclear hormone receptor, nhr-25, cause sensitivity to distinct, but overlapping stresses. [electronic resource]
Producer: 20141107Description: e92552 p. digitalISSN:- 1932-6203
- Animals
- Animals, Genetically Modified
- Antimicrobial Cationic Peptides -- genetics
- Caenorhabditis elegans -- genetics
- Coenzyme A Ligases -- deficiency
- DNA-Binding Proteins -- deficiency
- Gene Expression Regulation
- Gene Knockout Techniques
- Genetic Association Studies
- Longevity -- genetics
- Mutation
- Phenotype
- RNA Interference
- Stress, Physiological
- Transcription Factors -- deficiency
- Transcriptome
- Long-Chain-Fatty-Acid-CoA Ligase
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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