Absence of interleukin-17 receptor a signaling prevents autoimmune inflammation of the joint and leads to a Th2-like phenotype in collagen-induced arthritis. [electronic resource]
Producer: 20140527Description: 340-9 p. digitalISSN:- 2326-5205
- Animals
- Arthritis, Experimental -- pathology
- Autoimmune Diseases -- physiopathology
- CD4-Positive T-Lymphocytes -- metabolism
- Disease Models, Animal
- Inflammation -- physiopathology
- Interleukin-17 -- metabolism
- Interleukin-23 Subunit p19 -- deficiency
- Interleukin-4 -- metabolism
- Joints -- pathology
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Phenotype
- Plasma Cells -- pathology
- Receptors, Interleukin-17 -- deficiency
- Severity of Illness Index
- Signal Transduction -- physiology
- Th2 Cells -- pathology
No physical items for this record
Publication Type: Journal Article
There are no comments on this title.
Log in to your account to post a comment.