p53 inactivation decreases dependence on estrogen/ERK signalling for proliferation but promotes EMT and susceptility to 3-bromopyruvate in ERα+ breast cancer MCF-7 cells. [electronic resource]
Producer: 20140422Description: 169-77 p. digitalISSN:- 1873-2968
- Breast Neoplasms -- drug therapy
- Cell Proliferation -- drug effects
- Epithelial-Mesenchymal Transition -- drug effects
- Estrogen Receptor alpha -- biosynthesis
- Female
- Genes, p53 -- drug effects
- Humans
- MAP Kinase Signaling System -- drug effects
- MCF-7 Cells
- Pyruvate Dehydrogenase Complex -- antagonists & inhibitors
- Pyruvates -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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