Induced overexpression of phospholemman S68E mutant improves cardiac contractility and mortality after ischemia-reperfusion. [electronic resource]
Producer: 20140526Description: H1066-77 p. digitalISSN:- 1522-1539
- Adrenergic beta-Agonists -- pharmacology
- Animals
- Calcium Channels, L-Type -- metabolism
- Calcium Signaling
- Cardiotonic Agents -- pharmacology
- Disease Models, Animal
- Male
- Membrane Proteins -- genetics
- Mice
- Mice, Transgenic
- Mutation
- Myocardial Contraction -- drug effects
- Myocardial Infarction -- drug therapy
- Myocardial Reperfusion Injury -- drug therapy
- Phosphoproteins -- genetics
- Recovery of Function
- Ryanodine Receptor Calcium Release Channel -- metabolism
- Sarcoplasmic Reticulum -- metabolism
- Sarcoplasmic Reticulum Calcium-Transporting ATPases -- metabolism
- Sodium-Calcium Exchanger -- metabolism
- Sodium-Potassium-Exchanging ATPase -- metabolism
- Stroke Volume
- Up-Regulation
- Ventricular Dysfunction, Left -- metabolism
- Ventricular Function, Left -- drug effects
- Ventricular Pressure
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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