Ultraviolet radiation-induced inflammation activates β-catenin signaling in mouse skin and skin tumors. [electronic resource]
Producer: 20141126Description: 1199-206 p. digitalISSN:- 1791-2423
- Animals
- Casein Kinase I -- biosynthesis
- Cyclin D1 -- biosynthesis
- Cyclin D2 -- biosynthesis
- Cyclooxygenase 2 -- biosynthesis
- Cyclooxygenase 2 Inhibitors -- pharmacology
- Diet, High-Fat
- Female
- Glycogen Synthase Kinase 3 -- biosynthesis
- Glycogen Synthase Kinase 3 beta
- Indomethacin -- pharmacology
- Inflammation
- Matrix Metalloproteinase 2 -- biosynthesis
- Matrix Metalloproteinase 9 -- biosynthesis
- Mice
- Mice, Inbred C3H
- Mice, Knockout
- Prostaglandins E -- antagonists & inhibitors
- Proto-Oncogene Proteins c-myc -- biosynthesis
- Receptors, Prostaglandin E, EP2 Subtype -- biosynthesis
- Receptors, Prostaglandin E, EP4 Subtype -- biosynthesis
- Signal Transduction
- Skin -- immunology
- Skin Neoplasms -- immunology
- Ultraviolet Rays -- adverse effects
- beta Catenin -- biosynthesis
No physical items for this record
Publication Type: Journal Article
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